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Juerg Feldmann

Fortiori Design LLC
Posts: 1,530
When we  go  through the started  discussion  with load 1  - 4    where we  are stuck  for the moment in load  one  you will see some potential indication what we  see in your  data collections here. Here  just in short the   load discussion  graph thb smo2  excel l1 - l4.jpg

Now  when  you look your    graph  you can see, that in the short warm up you increase SmO2  nicely  as  an indication of better  saturation  and  more intake of O2  than  actual;ly needed  at that  load.
 . Interesting is  the relative minimal increase in tHb  and just short before  you stepped  of there  could have been a  small trend in increase in tHb.
. Than  at the   220  load you can see a  slightly but steady increase in SmO2  and    as  well in tHb.

 This  in many cases is  an indication , that  your body  was able to afford  to  sent  more blood as well as   in that blood good  saturated Hb  to  a non involved muscle.. This would indicate  , that 220 wattage is  for your   a  load  but not a  critical load  and you could go  most likley  for  a nice  duration.
. In fact  this  would be most likely  for  us in the STEI   intensity  , meaning , that   you are not  yet reached  any system  to a critical level of a limitation, just  may be nipping on it.
  Once you see a    flat SmO2  and a flat  tHb  you are  somewhere close  but not over it. Once yous see  in an  non involved muscle a change you  now know, that  some of the vital delivery system    have reached a critical intensity  an you simply try to " survive" by   regulating  in the   vascular  region for your loco motor  muscles the    priority of  O2  supply  and as  such as  well the   blood flow.  This is what  we mean  with  pyramid  of  Energy  supply.

hirarchye of O2.jpg

 This may make  now  more sense  for many. Now  this is  a  great " theory " but  as  so often before  we make a simple  picture  we   do some cases studies and than hope and search  for some back up  from other groups. . Here is a nice one    first. 

A method for assessing heterogeneity of blood flow and metabolism in exercising normal human muscle by near infrared spectroscopy

IoannisVogiatzis, HelmutHabazettl, ZafeirisLouvaris, VasileiosAndrianopoulos, HarriethWagner, Spyros G.Zakynthinos, Peter D.Wagner DOI:


Heterogeneity in the distribution of both blood flow and O2 consumption (VO2) has not been assessed by NIRS in exercising normal human muscle. We used NIRS to measure the regional distribution of blood flow and VO2 in six trained cyclists at rest and during constant load exercise (unloaded pedaling, 20%, 50% and 80% of peak watts) in both normoxia and hypoxia (FIO2=0.12). Over six optodes over the upper, middle and lower vastus lateralis, we recorded: a) indocyanine green dye inflow after intravenous injection to measure blood flow (Q), and b) fractional tissue O2 saturation (StO2) to estimate local VO2/Q ratios. Varying both exercise intensity and FIO2 provided a (directly measured) femoral venous O2 saturation (SfvO2) range from about 10 to 70%, and a correspondingly wide range in StO2. Mean Q-weighted StO2 over the 6 optodes related linearly to SfvO2 in each subject. We used this relationship to compute local muscle venous blood O2 saturation from StO2 recorded at each optode, from which local VO2/Q ratios could be calculated by the Fick principle. Multiplying regional VO2/Q by Q yielded the corresponding local VO2. While six opt odes along only in one muscle may not fully capture the extent of heterogeneity, relative dispersion of both Q and VO2 was about 0.4 under all conditions, while that for VO2/Q was minimal (only about 0.1), indicating in fit subjects: a) a strong capacity to regulate blood flow according to regional metabolic need and b) a likely minimal impact of heterogeneity on muscle O2 availability.


Here a very short discussion s ection  form an interesting  wokr done  in Japan.


There was no significant difference between MBP at 50 watts and that at rest. Since cardiac output

increases soon after the start of exercise, this unchanged MBP suggests vasodilatation in active muscles. After

ventilatory threshold (150 watts), the oxygenation level decreased. Since oxygenation level is a balance between

oxygen supply and since oxygen consumption is assumed to be constant in inactive muscle, the decrease in oxygen

level reflects a decrease in oxygen supply (Ogata 

et al.

2002). At this power output, mean BP showed a higher level. Therefore, vasoconstriction may occur in the inactive muscle. 

In  other words. When we reach a  critical  intensity, where we  can't afford  to  " waste" or  sent   O2  and blood  to  noninvolved muscels  the body will   react accordingly.
. So   using a noninvolved muscle in an endurnace   activity but as well during a strength  workout  or intervall can give you some fast and easy feedback, whether we reached  a  " delivery  limitation  of blood  and of  O2   from  the   vital  organs like  Cardiac  system  or  respiratroy system.

 This  would go into the same  thoughts of  reflex  reactions.
  Here the  full story in a picture  and now  for many again there may be a   better idea  why we use   a MOXY on a noninvolved muscle. It gives  you a relative fast feedback, whether the limiter is  in one of the  main delivery systems   or  whether we  may have more a  local  limitation.
ecgm pic.jpg 

  ecgm  2.jpg 












2 increased linearly after

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