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Juerg Feldmann

Fortiori Design LLC
Posts: 1,530
Here a beautiful IPAHD sent to me from Slovenia great job , smart person easy followed our ideas on the forum and produced an absolutely perfect IPAHD and combine it with his ideas of VO2  to come up with an incredible nice  result.
 Future MOXY users can do that in many different ways. Your first test ( cvs file ) can be sent to and we  try to tell you what you did and you can see when you  need some initial help.  Here to enjoy.
 Thanks so much and enjoyed your VO2  files you sent with it. Look in your mail for responds and closer details to this great assessment Juerg

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Juerg Feldmann

Fortiori Design LLC
Posts: 1,530
One  of many questions I get as well from this regular reader and coach is the question, :
 Why do we aim for a 5 min step or even longer like the IPAHD 5/1/5 protocol.
 There are many different reasons and I like to start with one of the more important reason the slow VO2 component, as well as the problem, that VO2 testing tackles  place  at the mouth so we have a "double " lag of results of VO2.
 This is, where it  is super interesting to have a live info of SmO2  and you can see the lag time as well as the need to have longer steps for interpretation and findings of limiter and compensator.
 As mentioned before the VO2 is much different respectively the SmO2  ( deoxygenation component ) is much more pronounced in short  intense loads , rather than in long all out   VO2 max tests. In fact in many cases we will see the highest VO2  after the end of a VO2 max test.
 Here a great   info on this   justification of 5/1/5 

Oxygen uptake dynamics: from muscle to mouth--an introduction to the symposium.


School of Sport and Health Sciences, University of Exeter, St. Luke's Campus,


The purpose of this paper is to provide an introduction to the study of oxygen uptake (VO(2)) dynamics or kinetics. Following the onset of exercise, both muscle and pulmonary VO(2) rise in a near-exponential fashion towards the anticipated "steady-state" VO(2) demand. However, it can take 2-4 min, or even longer at higher work rates, before this steady state is attained. Slow VO(2) kinetics increase the so-called O(2) deficit and obligate a greater contribution from anaerobic mechanisms of ATP production (involving the breakdown of muscle high energy phosphates and lactate production from glycogen) to meet the ATP requirement of the exercise task. A primary goal in this area of research is therefore to elucidate the physiological mechanisms which control and/or limit the rate at which muscle VO(2) increases following the onset of exercise. At higher intensities of exercise, a continued increase in both muscle and pulmonary VO(2) is observed with time despite the external work rate remaining constant. This continued rise in VO(2), beyond the anticipated steady-state requirement for the work rate, has been termed the VO(2) "slow component," and establishing the mechanistic basis for this phenomenon is another important goal of research in this field. This paper provides an overview of some of the factors which might contribute to both the fundamental and slow phases of the VO(2) kinetics and, in so doing, provides general background material for the more specific papers that follow.

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