Again the question on how do they relate with each other.
True they will relate n one or the other way, as all look for some similar information. This is to look
at intensities, where the body may start to look for help in the ATP maintenance besides the manly o2 supported metabolic options..
The question is, what motivates us to look for that.
The second question is, whether this info to find "aerobic" and "anaerobic" intensity is useful and for what.
, As well, whether all the different ideas give us enough information to get feed backs on why this " magical" threshold may have changed over night.
When we look on the base idea of survival, than we may agree, that to survive we can give up a lot of sections in our body without a problem on the survival rate.
Reducing O2 supply to many body areas like legs and arms will still allow us to survive.
So when we push our total body to a limitation, than we may in fact see the first reactions of a potential O2 delivery problem in our extremities. The main reaction in a very active O2 sucking extremity followed by body parts muscles not heavily involved in the activity and than we may see or feel a reaction in more vital systems.
So no wonder, that MOXY may give us the first time since many tools for testing got developed a direct window into this reaction as we look at directly at the origin of potential change.
. So changes in the working muscle all create some metabolic reactions. The integration of O2 independent ATP production will create substances like H + , CO2, and more and as such we will have some additional reaction seen in respiratory respond and or in changes in blood reactions ( lactate )
So the question is, whether the different threshold ideas may match up or at least may be close.
From the idea the direct information should be closer related to each other than the indirect information's.
So respiratory changes VT ( depending n Limiter) and MOXY reactions both direct test may be close to each other compared to lactate and or other indirect information's.
If the limitation is more from a central point of vie ( respiratory limitation ) than gas exchange and SmO2 will be closely related.
If the limitation is extremity , than respiration can be a compensator and the connection of SmO2 trend and gas exchange will be different.
This is as well the reason why in some cases VT and LT are closely related and in other cases they are completely off ( all depends on who limits and increases H + ) and who compensates and is able to control H + somewhat longer..
So as usual when we have this ideas we either wait for a study to back us up or we search for a study who was already done.
Here is an interesting one supporting the idea of using MOXY for individual training intensities.
Noninvasive determination of exercise-induced hydrodgen ion threshold through direct optical measurement
1. Babs R. Soller1,
2. Ye Yang1,
3. Stuart M. C. Lee2,
4. Cassie Wilson3, and
5. R. Donald Hagan4
+ Author Affiliations
1. 1Department of Anesthesiology, University of Massachusetts Medical School, Worcester, Massachusetts; and 2Wyle Laboratories, 3JES Tech, and 4National Aeronautic and Space Administration Johnson Space Center, Houston, Texas
1. Address for reprint requests and other correspondence: B. R. Soller, Dept. of Anesthesiology, Univ. of Massachusetts Medical School, 55 Lake Ave North, Worcester, MA 01655 (e-mail: firstname.lastname@example.org)
Submitted 7 August 2007.
Accepted 14 December 2007.
The intensity of exercise above which oxygen uptake (V̇o2) does not account for all of the required energy to perform work has been associated with lactate accumulation in the blood (lactate threshold, LT) and elevated carbon dioxide output (gas exchange threshold). An increase in hydrogen ion concentration ([H+]) is approximately concurrent with elevation of blood lactate and CO2 output during exercise. Near-infrared spectra (NIRS) and invasive interstitial fluid pH (pHm) were measured in the flexor digitorum profundus during handgrip exercise to produce a mathematical model relating the two measures with an estimated error of 0.035 pH units. This NIRS pHm model was subsequently applied to spectra collected from the vastus lateralis of 10 subjects performing an incremental-intensity cycle protocol. Muscle oxygen saturation (SmO2) was also calculated from spectra. We hypothesized that a H+ threshold could be identified for these subjects and that it would be different from but correlated with the LT. Lactate, gas exchange, SmO2, and H+ thresholds were determined as a function of V̇o2 using bilinear regression. LT was significantly different from both the gas exchange threshold (Δ = 0.27 ± 0.29 l/min) and H+ threshold (Δ = 0.29 ± 0.23 l/min), but the gas exchange threshold was not significantly different from the H+ threshold (Δ = 0.00 ± 0.38 l/min). This initial study demonstrates the feasibility of noninvasive pHm estimations, the determination of H+ threshold, and the relationship between H+ and classical metabolic thresholds during incremental exercise.
Att 1 is a 5 min step test MOXY on vastus lat and one moxy on delta pars acromialis as a bike test.
look at the tHb respond ( Trendline average ) at the end of the test Look at as well at the polynominal trend line. att 2
SmO2 drop in an inactive muscels and tHb drop would indicate a centrally controlled reduction in blood flow ( vasoconstriction) paired with a reduction in O2 delivery in this non essential area.
SmO2 drop with out activity would rather indicate a lower delivery than a higher utilization att 3 see red vastus lat and green delta trend in SmO2
att 4 polynominal SmO2.
Next up I will shwo you how we use "delivery " trends (tHb ) and utilization tends ( SmO2 ) to find individual "zoning" to desige an individual training to stimmulate the limiter and or stimmulate the delivery or the utilization systems.