Development Team Member
Registered: 1481997388 Posts: 59
Say we do some set of intervals with a regular warm up.
We get some SmO2/Thb graph showing fairly steep muscle desaturation and (hopefully) recovery. A perfect curve may look like this: Now let's speculate that we modify our training session and do the following: 1. Case 1: We "pre-load" the muscle so that lactate value at the beginning of the interval is large. Assume we can do this without stressing cardiac/respiratory. 2. Case 2: We do a very long "easy" session (so, cardiac/respiratory/muscles had a fair amount of stress, but they are not completely shot). Now we perform intervals. How would SmO2/Thb graphs look like for Case 1 and Case 2 compared to the ideal case above? I am of course aware that especially in Case 2, the graph will almost certainly depend on on what your limiter/compensator is. Related question: is there a big physiological benefit of preloading some system? It seems to me that the main effects could be: a) compensator training (ie, limiter may have reached a limit, so compensator has taken over) b) limiter training It seems that the case for limiter training here is pretty weak since e have already weakened already weak system.
Development Team Member
Registered: 1380484167 Posts: 1,501
Great points and great thoughts.
Here some ideas to it : 1. This is the weakness, when you only take SmO2. You need to learn to intergate tHb as there are different reasons why SmO2 drops. a) Delivery limitation due to occlusion reaction in the load. b) still available blood flow c) outflow restriction due to the load So any of this three will create a different outcome to your questions. The pre load of lactate is a fascinating options for a whole set of physiological stimulations. Here an old case study long time back done with Portamon. with a world class athlete where we were looking at options of pre loading lactate with different interventions. Top green is TSI % in the bottom graph with red and blue green is tHb . The first lactate number is immediately taken after the interval , the second number is taken 3 min later. The goal was to keep or even increase lactate during the rest period . Practical use as preparation for certain events or during certain races. That is the idea for performance improvement. What I use it much more is for physiological stimulation of MCT 1 and MCT 4 proteins. Now as usual we where at that time not only looking at NIRS and lactate in cases like this but as well how the cardiac and or the respiration would react. Below three loads as you can see, 2 are manipulated differently and the interesting part is , that we in fact are able to manipulate cardiac reactions as well. Top left is HR to right is SV Bottom left green is HR x SV = CO and bottom left is EF % ( ejection fraction %) Now the next part to combine to get a full picture or nearly a full picture is to look at respiratory manipulation and SmO2 ( tHb ) reactions . Now the below case studies where done by Nick Mclean now in Australia for his masters. Nick was a local student who was a part of our grade 10 - m12 exercise physiological play time. The idea was Pe load of different physiological systems to create a specif stimulus for a specific physiological target. Examples are specific preloads for a follow up right ventricular load. Or for MCT 1 stimulation, or for intermuscular stimulation or for blood volume shift from one body are to another.. This steps are very helpful where there is time restriction for example or where we have to avoid overloading certain systems like in COPD clients or in cardiac clients and others like restriction of load due to post ops restriction. Here a manipulation in recovery and workout length using a specific respiratory device. Always the same load but different interventions which as you can see changed the duration off the performance.