Fortiori Design LLC
Registered: 1355349061 Posts: 1,530
One of the main reason, why we have only a few published NIRS studies with strength is the simple fact, that you may have a very big advantage if you can use NIRS on a muscle chain rather than on a single muscle.
The cost involved to have even just a single moxy site to test was huge so the coaches and centers simply could not afford the testing. As well the problem with actually using it practically in a gym. Well with MOXY both problems are solved. When you look the cost of a great " traditional NIRS to the cost of MOXY , than teh ability to see SmO2 on a wrist watch compared to the whole equipment you may use with the great classical equipment shows you why MOXY is the big runner form the upcoming season for coaches and athletes. Here another great summary who shows why NIRS should be used but they still have the problem and miss the information, that MOXY is on the market for anybody to buy. I just had a discussion with a " leading' Ph.D guy for NIRS and specialist who had no information what's how ever, that you can buy MOXY online. Here to read: A brief review of the use of near infrared spectroscopy with particular interest in resistance exercise. Source
Departmento de Educação Física, Universidade Gama Filho, Rio de Janeiro, Brazil.
There is growing interest in resistance training, but many aspects related to this type of exercise are still not fully understood. Performance varies substantially depending on how resistance training variables are manipulated. Fatigue is a complex phenomenon usually attributed to central (neuronal) and/or peripheral (muscular) origin. Cerebral oxygenation may be associated with the decision to stop exercise, and muscle oxygenation may be related to resistance training responses. Near infrared spectroscopy (NIRS) is a non-invasive optical technique used to monitor cerebral and muscle oxygenation levels. The purpose of this review is to briefly describe the NIRS technique, validation and reliability, and its application in resistance exercise. NIRS-measured oxygenation in cerebral tissue has been validated against magnetic resonance imaging during motor tasks. In muscle tissue, NIRS-measured oxygenation was shown to be highly related to venous oxygen saturation and muscle oxidative rate was closely related to phosphocreatine resynthesis, measured by (31)P-magnetic resonance spectroscopy after exercise. The test-retest reliability of cerebral and muscle NIRS measurements have been established under a variety of experimental conditions, including static and dynamic exercise. Although NIRS has been used extensively to evaluate muscle oxygenation levels during aerobic exercise, only four studies have used this technique to examine these changes during typical resistance training exercises. Muscle oxygenation was influenced by different resistance exercise protocols depending on the load or duration of exercise, the number of sets and the muscle being monitored. NIRS is a promising, non-invasive technique that can be used to evaluate cerebral and muscle oxygenation levels simultaneously during exercise, thereby improving our understanding of the mechanisms influencing performance and fatigue.
PMID: 17595156 [PubMed - indexed for MEDLINE]
Fortiori Design LLC
Registered: 1355349061 Posts: 1,530
What do we do with strength and MOXY.
We are in the midst of preparing a starter e book info on MOXY and strength workouts. The critical situation is to not jump brutally into the existing ideas but rather combine many great existing ideas by slowly introducing MOXY into the tools for training intensity a and recover length control. We use MOXY ( NIRS since many years fro strength and we may simply think very different than what most of us are used with strength as well as with endurance wo4krouts. I like to show here a very short idea on how a strength workout looks for my clients. a) elderly client with know joint problems. 1. "lubrication" active ROM through the motions of the upcoming workouts with control of increasing SmO2 and tHb. It will take often about 5 - 10 min till we reach a common level or a "plateau" Set 1. Motion till we reach a low SmO2 and than hold the SmO2 for 5 seconds low by keeping going. The time I like to reach the low SmO2 is in 10 - 15 seconds from the start. Than rest and re oxygenation back to start level. followed by second Set same weight and goal to reach the same SmO2 low level or lower and hold low level for 5 seconds. rest as before and look at time changes. Now how many sets. a) either till he or she can't deoxygenate anymore down to the same level or reoxygenate back to the start level in a decent time. Often the sets will be achieved somewhere around 3 - 8 sets. . Subjectively : If a client can reach 10 sets and still all is well I increase the weight next time so he " fails somewhere around 5 sets. Why the plateau. Experience and results. If I stop by low SmO2 without a plateau I see much less progress if any. If I increase SmO2 plateau longer to 10 - 15 seconds than this age group has severe DOMS for a few days and due to the arthritis get stiffer in the joint. I like that they can do this workout 2 - 3 times per week or 1 day work 2 days off. Possibly not optimal for strenght gain but needed to maintain ROM in joints. If I deoxygenate and keep ;plateau for 10 - 15 seconds I can do 2 workouts with work 3 - 4 days rest for that muscle group and gain more performance . Possibly due to the stimulus the Hypoxia creates. Now this is all experience as we do not have any scientific studies done with hormonal control and other options. BUT as NIRS is getting more popular researcher try to look why this is the case. And here a nice paper backing our years of ideas up in 2013 . Significant Molecular and Systemic Adaptations after Repeated Sprint Training in Hypoxia Published: Feb 20, 2013 DOI: 10.1371/journal.pone.0056522 Abstract
While intermittent hypoxic training (IHT) has been reported to evoke cellular responses via hypoxia inducible factors (HIFs) but without substantial performance benefits in endurance athletes, we hypothesized that repeated sprint training in hypoxia could enhance repeated sprint ability (RSA) performed in normoxia via improved glycolysis and O
utilization. 40 trained subjects completed 8 cycling repeated sprint sessions in hypoxia (RSH, 3000 m) or normoxia (RSN, 485 m). Before (Pre-) and after (Post-) training, muscular levels of selected mRNAs were analyzed from resting muscle biopsies and RSA tested until exhaustion (10-s sprint, work-to-rest ratio 1:2) with muscle perfusion assessed by near-infrared spectroscopy. From Pre- to Post-, the average power output of all sprints in RSA was increased (p<0.01) to the same extent (6% vs 7%, NS) in RSH and in RSN but the number of sprints to exhaustion was increased in RSH (9.4±4.8 vs. 13.0±6.2 sprints, p<0.01) but not in RSN (9.3±4.2 vs. 8.9±3.5). mRNA concentrations of HIF-1α (+55%), carbonic anhydrase III (+35%) and monocarboxylate transporter-4 (+20%) were augmented (p<0.05) whereas mitochondrial transcription factor A (−40%), peroxisome proliferator-activated receptor gamma coactivator 1α (−23%) and monocarboxylate transporter-1 (−36%) were decreased (p<0.01) in RSH only. Besides, the changes in total hemoglobin variations (Δ[tHb]) during sprints throughout RSA test increased to a greater extent (p<0.01) in RSH. Our findings show larger improvement in repeated sprint performance in RSH than in RSN with significant molecular adaptations and larger blood perfusion variations in active muscles. 2