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Juerg Feldmann

Fortiori Design LLC
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Posts: 1,530
 #1 
You will figure out  what they  where asking  for.
A:
No, in any endurance sport  ( in theory where more than 60 % of the total muscle mass is involved) the location of the MOXY  is not important  for a workout.
 The trend is , what you look  for.
  So if we   believe the idea  of a central  control of O2  is  real , we should have no problem with any location  .
  We  can push it so far, that we  not even need a muscle  which is involved directly in the sport.
 Example  cycling :
 I  can have a MOXY on a leg muscle as the involved  muscle  and can have a MOXY  for example on  the rectus  abdominalis  as  an expiration muscle.
 I  can do a workout  and will have the same trend information.

 In fact  what will happen is :
  If  we have  a muscle , which is not involved or very minimal involved , we have the advantaged,  if not making a 5/1/5  to see, whether we  actually have an optimal  " warm up" as in the not involved muscles  we have a much better reaction on additional blood flow  due to increase in CO  . VE  and  capillarisation.
 IN an involved muscle we  have always the question between muscle tension  due to contraction  and real increase in blood flow.  see the specific  workout   from this athlete  and you tell me  which muscle was the leg muscle  and which one  was the respiratory  abdominal muscle.

 The interesting  observation is:
  If  we  start to run into  "trouble " in the leg muscle;s  with the delivery system  we  will first , before we  shut    recruitment or  blood flow down  search  for  an additional compensation.
 So if a  muscle , which is not really heavily involved in the task still has a lot  of  blood  flowing  through we will see a  drop in tHb  at the same time  as we see a drop in tHb in the working muscle.  The body  in the not involved muscle will create a vasoconstriction to reduce blood flow.  As the muscle is not involved  we know the drop is due to   vasoconstriction and not like in the working muscle, where muscle tension could be a   reason for a dropping tHb.
 When looking at SmO2. If a muslel is not involved in  action  than we do not  need or see a drop in SmO2  due  to utilization. In an inactive muscle a drop in SmO2  is an indication over a   controle system, which  reduces  delivery  to the non working muscle  rather than  actually using  it so we  have  an additional way of getting O2  to the working muscels.
  See this  fun example . look at the  SmO2    see, which  one may be  active muscle and  which  one is  the non working muscle.  ( exception here is that the rectus will start  to do some work  if  respiration is in  trouble,.
  Here just  for fun a  small inside  view  in what  you  can expect on informatiosn in our MOXY  seminars  or when we look through test  you sent to us  for interpretation. Over time you will learn  and see more  and more  on your own.

Attached Images
Click image for larger version - Name: thb_involved_and_not_involved..jpg, Views: 15, Size: 62.34 KB  Click image for larger version - Name: SmO2_load_and_HR_leg_inv.jpg, Views: 15, Size: 74.26 KB  Click image for larger version - Name: uninvolved_with_load.jpg, Views: 14, Size: 58.72 KB 

Juerg Feldmann

Fortiori Design LLC
Registered:
Posts: 1,530
 #2 
Is there  some  "accepted " studies   on what I postulate  above.
Three mails in 15 min with this   question.
  So    I did some searching and here a very short  answer  from one of the studies  I found  from Japan.
  "

"Near-infrared spectroscopy (NIRS) is a new method by which oxygenation level in the tissue can be

determined. We have examined oxygenation level in inactive muscle during exercise using NIRS (Ogata

 

et al.

2002). We found that the oxygenation level did not change at low exercise intensity but decreased at high

exercise intensity after about two minutes of exercise. Since the oxygenation level is determined by a balance of

oxygen supply and since oxygen consumption can be assumed to be constant in inactive muscle, the decrease in

oxygenation level in inactive muscle can be assumed to be due to the decrease in oxygen supply. Moreover, it has

been pointed out that oxygenation kinetics in inactive muscle during exercise is similar to the characteristics of

activation of sympathetic nervous system due to exercise. This suggests that attenuation of oxygen supply is due to

vasoconstriction in inactive muscle during exercise.


 What we  add is the readings  of Thb  which they never used  and most NIRS studies never look at.  If the vasoconstriction takes place  as pointed  out  than you see what we  show in one of  our many case studies.

 

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