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Juerg Feldmann

Fortiori Design LLC
Registered:
Posts: 1,530
 #1 
Congratulation to Dominique, one of the many quite  but incredible out of the BOX thinking coaches. Dominique is one of the extraordinary brains in Swimming  and in the medical field. challenging her own ideas  and thinking outside the BOX.
We  are privileged have met  Dominique in one of our MOXY seminar in Boulder   and her open     mind  and her incredible ability to  combine and adjust  physiological critical questions  with practical application has created a  new  Commonwealth record  for Canada in swimming. Some readers  may recall  her  worlds  first   assessment with NIRS in the water despite some  resistance  she has to overcome in her  own sport  against traditional ideas  and  brains.
 Dominique great work  and it was  fun to see how  you intergate3d  physiological information into a sport  overloaded  with traditional ideas of training . A  sport, where   numbers  of reps  and distance is the  traditional guide of  intensity rather than individual  information of the athletes  ability  to delivery  an utilize energy.
 Here   a very small insight  view in a  s super intelligent   training  concepts.

Here  some  comments  as  an insight view into a  genius coaching brain.

"

From xy’s graph, I can tell that even the rest is shorter during the first set (12x50 pull on 1'30), her SmO2 is coming up fairly at the same level.  What is interesting is even she is wearing paddle (during the second set), her SmO2 doesn't go lower.  We like to use those device to be able to build some power by developing some strength (force).  Either she didn't work hard on it or for her, it doesn't work the waywe thought. During the last set, she had to swim fast.  Her time were good even she was not kicking much.  We can tell by the lower SmO2 values that she did work harder then during the first set but not that much because there is not much difference between having a pull (between your thighs) and ...."


Here a graphic  view  of one of the individual workouts  and the   thoughts on what is going on.

3 blocks thb smo2.jpg 
 And  here a very close look on what in this particular case  the limitation  was or is  and where the focus  had to be  made.
D K close look  last set.jpg

You see what creates a  potential limitation  an this here is  in the last set.
 The  race  she  won was  coming  " from  behind" , which  was the comment of the   Pro's on the TV. Reality was, that she was the only one  which maintained the  speed.  The same  was true  in the gold medal  by the men's 400 m  , where the only swimmer maintaining the pace  was the winner. The  Problem is, that in the graph above the critical information would be if tHb  would plateau   and SmO2  still would  drop :
  Why ??

 On the other side in a  sport like ice hockey  you would like to see tHb   plateau during a shift  and not what we see here.
:  Why ? Here a  closer look  at a NHL player  with the needed  load to create the tHb respond  for one shift.

aother case  occl  l 1.jpg


And here  just for fun for critical brains a great feedback from Dominique  from a work out where the athlete  had to change  intensity  and you can see how the coaches  idea  shows  up  as a perfect feedback  from the athletes  muscle  information

Question  where and what  shows the change in  i8ntensity  and what has changed. ?


chnage  from  free flwo to occ t.jpg

ryinc

Development Team Member
Registered:
Posts: 369
 #2 
Thanks for the interesting data on this case study.

I recall in one of the other case studies discussing venous occlusions in cycling. It was discussed there that venous occlusion trends in cycling would normally not show up as a drop in tHb on rest as it would in say a venous occlusion in an exercise with much higher force (e.g. weight lifting say). In other words, a venous occlusion trend in cycling might actually simply show up as a very small drop, flat or even slow increase in tHb at the end of the load.

Now, i don't know anything about swimming physiology but i would have imagined that the compression of muscles in swimming would be even lighter than in cycling and so even less likely to see a venous occlusion trend this pronounced

Any comments on what muscle was being measured here and why it shows up so obviously in swimming but not cycling? 
juergfeldmann

Development Team Member
Registered:
Posts: 1,501
 #3 
In swimming we look  often  on lattisimus  dorsi or pectoral muscles in upper body  and on hip  quadriceps  rectus  femoris  and  or   hamstrings in legs.

Now, i don't know anything about swimming physiology but i would have imagined that the compression of muscles in swimming would be even lighter than in cycling and so even less likely to see a venous occlusion trend this pronounced

 I was actually  hoping  some of the cycling community would  jump in here on this interesting topic.

So  for MOXY owner  here a  very simple case  study you can do..
 You can do  all on dry land  but if  you  have problems  to believe the  information  you can do on in the water.

 Fix a MOXY on the rectus femoris  of   your leg.
 Now  sit on a bike  and you can push in different angles like 12.00 clock  and than every  perhaps 30 degrees  as hard on the pedals  as you can do. Use your break  so we have an isometric  action. If  you  have a SEMG  add this  to the feedback.
Now  do a 1 leg  squatting after this.

 Now  do a   back or  front crawl motion  with the legs  as  hard  as you can.You can  either  sit on the ground or   on the back  and work  to  failure.

 Than look the SmO2  reactions in all three cases  and make your  conclusions.

Now here   an old  comparison of   rectus  femoris  activity of  2  world  class  MTB  athletes. The dates  are  from a Portamon study  and you can actually  figure out  the RPM  they where  pushing  it that stage  with same  wattage  for  both. Despite the fact , that they where in that years  very  close in  races  they have  completely different limiters.
 One  of them  surprised  us most . it is  the    actual    reaction  on blood flow or how  they  bike.
 The graph  shows  you tHb reaction  of both.
  We  than analyzed  it  closer  with   HHb and O2Hb reaction  and there was the difference.
 One  has a   situation where, when tHb goes sup it  is  due to a decompression reaction of the  RF  and when it goes s down  it is  due to a  compression   of the  muscle on the  blood vessels.
 The other athlete  had  a   situation where, when   tHb  increased  he  actually  created a venous occlusion and  when tHb  dropped  it was  an occlusion outflow. Optically  it looks  the same .
 So  as we  have limited  information looking  at SmO2 alone  so  do we have limited information looking tHb alone.

yama gk rpm tHb.JPG

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