Development Team Member
Registered: 1380484167 Posts: 1,501
Additional thoughts to Rynic respiratory Ac and a information.
If the respiration ( not a metaboreflex ) is the limitation so VE is a limitation , than we have a situation, where we may move towards hypercapnia meaning we accumulate more CO2 than we can get rid of. This means a limitation in H + balance as well and as such we have an increase in RF as a high CO2 tries to stimulate the respiratory system. But if the TV is limited than we only can increase RF and often see than a reduction in TV. ( think dead space and what that means.) So the increase in CO2 will show up systemic and we see now at the "VT 1" when we reach it o go above a very specific NIRS reaction when we stop . What do we see.? . On the other hand ,if the VT 1, ( same picture) is created due to a compensation by a great respiratory system, so we actually get easy rid of CO2 , in fact we could as an idea even go hypocapnic, than we see, when we stop at or above the "VT 1" what kind o a NIRS reaction.? . Now for lactate believers this will again give a very different picture. In the situation of a limiter the lactate values will be actually lower, as it is NOT the lactate who stops the performance, but the respiratory limiter. On the other hand if w can get rid of CO2 and therefor are able to balance H + longer and keep the O2 disscurve stable for longer we will see a higher lactate value,but despite or because of the high lactate we can as well go longer, as we have more and easier energy available (lactate) for ATP maintenance. So the contradiction for classical lactate user . A lower lactate will stop performance and a higher lactate will keep them longer going. The reason is the respiratory either limitation or compensating situation.