Fortiori Design LLC
Registered: 1355349061 Posts: 1,530
We are since many month in an ongoing discussion on this specific great idea.
Bio availability of O2. What it means is the situation, where we have O2 in the blood and on Hb and on Mb but out of what ever reason we can't get to it. It is not bio available and as such we may have lot's of O2 there but we will create a hypoxic situation. So this opnes a set of question. If w e have a normoxic situation with % of O2 , what do we have to do to get into a hypoxic ( O2 not available ) situation. what benefit do we get if we would create a hypoxic situation and what would happen if we create a hyperoxic situation. We can create this situation all in artificial ways with hypoxic tents or rooms and hyperoxic equipment. The studies we have form this can help us further. What we did mainly in the past is , that we used a SpO2 sensor and as such had feed backs on O2 situation which occur between lungs ( outside world ) and blood. BUT we have no information ( feedback ) what happens between blood and cell. So using an SpO2 sensor for hypoxic. workouts is a good first step but little help to our question, whether it is bio available. A much more interesting task is, whether we can create a normoxic ( for sure ) hypoxic for sure ) but as well a hyperoxic unsure ) situation in a natural way by simply=. learning to manipulate respiration. Here a first step into some of the possible answers. I got this incredible data collection sent form Prag. By Jiri and his out of the box working group. Here a small inside view. This are three clients they test in a step test option of 6 min each step. Some where just following their bodies lead, t some had to change in the midst of the 3 min the respiratory p[pattern from so called " normal " natural" breathing to a fast more intense breathing, which was called hyperventilation. , Whether it was hyper ventilation ( meaning the EtCO2 should drop as they breathe more than needed and as such would possible get hypocapnic ) was not in the task. They simply had to try to breath much more than they normally would do. The one question was, whether the change in respiration no mater what they would achieve would change the bio availability of the muscles and seen in a MOXY data collection. Than the question would be, whether the change can be explained by a change in O2 diss curve shift. And in this matter, whether the CO2 would influence the bio availability just locally in the working muscle or whether we would have some reactions as well in a non working muscle or a muscle , which would work in sever involvement of a respiratory work. So they used 2 MOXY's one on the vastus lateralis during the bike workout and one on the trapezius so upper trapezius on the same side. The regular reader out of the box think critical coach will be able to read an incredible amount out of just this info sheet here. You see dark green as SmO2 trend of vastus lateralis, light green as trapezius SmO2 trend and Grey as the EtCO2 tested at the mouth with a VO2 equipment. Have fun and you will see, where we can go with that.